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Rapamycin-induced small extracellular vesicles under GelMA scaffolds facilitate diabetic wound repair through accelerating angiogenesis and alleviating macrophage-mediated inflammation via PI3K/Akt signaling pathway

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Semaglutide and tirzepatide are "upstream" — they act on incretin receptors to influence appetite regulation, gastric emptying, and glucose-dependent insulin secretion

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