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Therefore, developing and validating biomarkers that directly indicate neuronal ferroptosis—such as specific oxidized phospholipid profiles, ferroptosis-related proteins (e.g., released HMGB1, modified GPX4), or their metabolic fragments—is crucial for enhancing clinical diagnostic and therapeutic monitoring capabilities
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476 Indeed, a specific deficit in the induction of enzymes of 1C metabolism leads to the accumulation of impaired naive T cells in aged mice, which could be rescued by the addition of products of 1C metabolism (formate and glycine) to the cells
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