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Allicin promotes functional recovery in ischemic stroke via glutathione peroxidase-1 activation of Src-Akt-Erk | Cell Death Discovery

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This activation reduces the inhibitory binding between Caveolin-1 and eNOS, allowing greater eNOS activation and nitric oxide production—with laboratory studies showing BPC-157 at 1 μg/ml increases NO production 1.35-fold compared to controls