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Thus endogenous dopamine, as well as exogenous treatment with levodopa (used in PD) may be a further source of oxidative stress that may worsen pathogenesis [71, 72]
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These observations fit prior work in Atm –/– mice demonstrating that treatment with antioxidants N-acetyl cysteine (NAC) or nitroxides (isoindoline nitroxide, CTMIO or Tempol) increased survival, delayed lymphoma onset, and reduced genomic instability 13,14,15,16,17,18,19,20
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