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however, storage only lasts up to 18 days.[3–6] The absorption of thiamine occurs in the duodenum by an active process and is converted to its active form thiamine pyrophosphate.[7,8] This conversion process of thiamine to its active form requires magnesium as a cofactor, and hence hypomagnesaemia can mimic thiamine deficiency.[6,9,10] The absorption process of thiamine is a carrier-mediated, rate-limiting process,[11] and alcohol inhibits this rate-limiting step by interfering with intestinal ATPase involved in the absorption of thiamine.[12,13] At the blood brain barrier (BBB), its transport occurs through both passive and active mechanisms[14] depending on the concentration of thiamine in the blood