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Pre-treatment of the PBMCs with antioxidants, tiron, L -histidine, and D -mannitol, reduced the observed number of lesions to the same level as the untreated sample, implicating superoxide, singlet oxygen, and hydroxyl radicals, respectively, in lesion formation
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The lack of a consistent correlation between GST polymorphisms and PD does not rule out a significant role for this enzyme in the etiology of PD, as there could be other regulatory influences on these proteins, including those controlling synthesis and breakdown
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