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FIGURE 1 Alternatively, the interaction of Keap1-Nrf2 can be disrupted by non-canonical mechanisms (Figure 1)

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Species differences in N -glucuronidation The highly selective non-covalent inhibitor of Bruton’s tyrosine kinase (BTK) pirtobrutinib is another drug mainly metabolised by CYP3A4 and direct glucuronidation, the latter via UGT1A8 and UGT1A9